Anxiety and Fish Oil
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The use of fish oils to mediate anxiety in a selected group of intelligent and healthy young people has suggested a positive correlation. The implications are that other groups, especially the elderly and individuals with complex disorders may also benefit. For over thirty years the relationships between stress and immune function have been explored and this is one of the latest papers published in the leading cross discipline journal Brain Behaviour and immunity.[1]
Fish Oils & Arthritis, What The Prescriber Needs to Know
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When faced with the question what do I need fish oils for and what dose should I be taking for the management of pain and stiffness, it is always handy to be able to refer to a reliable third party source. This open access paper describes just ‘what the prescriber needs to know’. This manuscript speaks directly to practitioners and patients who would like to experience fish oil benefits. It was published in 2006 by researchers from Royal Adelaide Hospital, Adelaide, Australia. It is worth downloading and keeping on your computer for ease of reference.
Dietary Reference Intakes for EFA’s
There is considerable interest in the impact of (n-3) long-chain PUFA in mitigating the morbidity and mortality caused by chronic diseases. In 2002, the Institute of Medicine concluded that insufficient data were available to define Dietary Reference Intakes (DRI) for eicosapentaenoic acid (EPA) or docosahexaenoic acid (DHA), noting only that EPA and DHA could contribute up to 10% toward meeting the Adequate Intake for alpha-linolenic acid. Since then, substantial new evidence has emerged supporting the need to reassess this recommendation.
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The immune system is prone to the same grave misfortunes as any defense system handling weapons: collateral damage that comes with the destruction of the enemy on one’s own territory and friendly fire due to mistaken identity. Whereas the collateral damage is the price we pay for clearance of infections, autoimmunity is a pathological process. Nevertheless, the effector mechanisms involved in both processes are the same. Whether environment can be a cause, a trigger or an amplifier of an autoimmune disease are questions that are being intensively investigated.
