Vitamin D and Parkinson’s Disease Correlations Identified
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Back in 2008 a team of researchers published a paper in the Archives of Neurology that exposed a striking incidence of Vitamin D deficiency in patients diagnosed with Parkinsons Disease.[1] They compared the Parkinson’s patients with healthy controls and patients with diagnosed Alzheimers disease. In both groups the Parkinson’s patients presented with reduced Vit D status compared to the others.
Oral Glutathione Equivalent to IV Therapy!
Michael Ash BSc DO ND F.DipION and Marty Jones PharmD review the changing face of glutathione and explore the acetylated form as an alternative to IV glutathione therapy.
Reduced glutathione also known as glutathione or GSH is found in all living systems.[1] Lowered tissue GSH levels have been observed in several disease conditions.[2] The restoration of cell GSH levels in a number of these conditions have proven to be beneficial. Thus, strategies to boost cell glutathione level are of marked therapeutic significance.
GSH is the smallest of the intracellular thiols (a compound that contains the functional group composed of a sulphur-hydrogen bond (-SH) hence its unpleasant smell when mercaptans are released) and its high donating electron capacity combined with dense intracellular concentration provides significant oxidative reducing capacity.[3]
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Folic Acid-Indication: Parkinson’s Disease (PD)
Research: In this study, researchers measured blood levels of total homocysteine ((t)Hcy), vitamin B(12) and folic acid in patients with Parkinson s disease (PD) and in age-matched controls, and searched for possible associations between these levels with smoking, alcohol consumption, L-DOPA treatment and disease duration in PD patients.
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The immune system is prone to the same grave misfortunes as any defense system handling weapons: collateral damage that comes with the destruction of the enemy on one’s own territory and friendly fire due to mistaken identity. Whereas the collateral damage is the price we pay for clearance of infections, autoimmunity is a pathological process. Nevertheless, the effector mechanisms involved in both processes are the same. Whether environment can be a cause, a trigger or an amplifier of an autoimmune disease are questions that are being intensively investigated.
